INT62992

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Context Info
Confidence 0.26
First Reported 1996
Last Reported 1996
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.60
Pain Relevance 0.30

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (COLQ)
COLQ (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 3 93.36 High High
Action potential 1 89.60 High High
ischemia 4 75.00 Quite High
action potential duration 1 71.20 Quite High
lidocaine 1 25.00 Low Low
Mexiletine 1 25.00 Low Low
antiarrhythmic agent 1 25.00 Low Low
Disease Link Frequency Relevance Heat
Accelerated Idioventricular Rhythm 1 96.28 Very High Very High Very High
Tachycardia 1 94.72 High High
Congenital Anomalies 2 76.80 Quite High
Cv Unclassified Under Development 4 75.00 Quite High
Myocardial Infarction 1 69.24 Quite High
Arrhythmias 2 Under Development 1 37.36 Quite Low
Ventricular Tachycardia 2 34.32 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
If the cause enhanced impulse generation is related to triggered activity, and since both EAD and DAD are dependent on calcium currents that can appear during a delayed repolarization, the therapeutic options are to shorten the repolarization phase through K-channel openers or Ca antagonists, or to suppress the inward currents directly responsible for the afterdepolarization with Ca blockers.
Regulation (dependent) of EAD associated with antagonist
1) Confidence 0.26 Published 1996 Journal Clin Cardiol Section Abstract Doc Link 8706374 Disease Relevance 0.60 Pain Relevance 0.30

General Comments

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