From wiki-pain
Revision as of 11:45, 20 September 2012 by Daniel (Talk | contribs)

(diff) ← Older revision | Latest revision (diff) | Newer revision → (diff)
Jump to: navigation, search
Context Info
Confidence 0.44
First Reported 1999
Last Reported 1999
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.74
Pain Relevance 0.80

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Gabbr2) cytoplasm (Gabbr2) signal transducer activity (Gabbr2)
Anatomy Link Frequency
sensory systems 2
Gabbr2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
gABA 8 100.00 Very High Very High Very High
dorsal root ganglion 1 97.52 Very High Very High Very High
Spinal cord 2 93.40 High High
Lasting pain 2 92.16 High High
Pain 3 77.28 Quite High
Antinociceptive 1 61.28 Quite High
nociceptor 2 25.00 Low Low
Disease Link Frequency Relevance Heat
Nociception 4 97.88 Very High Very High Very High
Ganglion Cysts 1 97.52 Very High Very High Very High
Pain 4 92.16 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These findings indicate an activity-dependent, differential regulation of GABA(B) R1 and R2 receptor gene expression in spinal sensory systems in response to chemogenic nociceptive activation, suggesting that GABA(B) receptor plasticity may play an important role in regulating the mediation, and perception, of chronic pain.
Regulation (regulation) of Gene_expression (expression) of R2 receptor gene in sensory systems associated with nociception, gaba and lasting pain
1) Confidence 0.44 Published 1999 Journal Neuropharmacology Section Abstract Doc Link 10587092 Disease Relevance 0.74 Pain Relevance 0.80

General Comments

This test has worked.

Personal tools