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Context Info
Confidence 0.41
First Reported 2001
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 1.80
Pain Relevance 0.56

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Bcl2a1a)
Anatomy Link Frequency
colon 1
microglia 1
Bcl2a1a (Mus musculus)
Pain Link Frequency Relevance Heat
primary somatosensory cortex 15 100.00 Very High Very High Very High
adenocard 43 99.96 Very High Very High Very High
cINOD 1 94.96 High High
agonist 6 15.92 Low Low
Nerve growth factor 5 11.52 Low Low
cytokine 9 5.00 Very Low Very Low Very Low
Inflammation 8 5.00 Very Low Very Low Very Low
ischemia 7 5.00 Very Low Very Low Very Low
Inflammatory mediators 4 5.00 Very Low Very Low Very Low
Endocannabinoid 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 10 100.00 Very High Very High Very High
Disease 16 99.58 Very High Very High Very High
Colon Cancer 1 99.30 Very High Very High Very High
Cancer 2 94.88 High High
INFLAMMATION 15 93.52 High High
Multiple Sclerosis 4 91.60 High High
Sprains And Strains 44 86.32 High High
Sclerosis 5 70.64 Quite High
Death 6 52.04 Quite High
Targeted Disruption 9 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Recent results from experiments designed to evaluate drug-induced apoptosis in colon cancer cells revealed that the levels of BCL-2-related apoptotic suppressor proteins were dramatically reduced compared with those of pro-apoptotic proteins.
Negative_regulation (reduced) of BCL-2-related in colon associated with colon cancer and apoptosis
1) Confidence 0.41 Published 2001 Journal Trends Endocrinol. Metab. Section Abstract Doc Link 11306332 Disease Relevance 0.77 Pain Relevance 0.09
A Procrustes superimposition of 7 landmarks across V1, S1, and A1 from each of 16 adult C57BL/6J mice and 15 adult DBA/2J mice is shown in Figure 5.
Negative_regulation (superimposition) of A1 associated with primary somatosensory cortex
2) Confidence 0.13 Published 2006 Journal BMC Neurosci Section Body Doc Link PMC1578583 Disease Relevance 0.37 Pain Relevance 0.13
The involvement of endogenous adenosine in the development of the disease is supported by the downregulation of A1 adenosine receptors in microglia of A1+/+ mice and by the beneficial effect of caffeine, which upregulates A1 receptors and reduces disease severity concomitantly [99].
Negative_regulation (downregulation) of A1 in microglia associated with adenocard and disease
3) Confidence 0.05 Published 2006 Journal Purinergic Signal Section Body Doc Link PMC2096753 Disease Relevance 0.66 Pain Relevance 0.34

General Comments

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