INT99298

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Context Info
Confidence 0.06
First Reported 2001
Last Reported 2005
Negated 0
Speculated 0
Reported most in Abstract
Documents 6
Total Number 6
Disease Relevance 2.94
Pain Relevance 1.54

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell adhesion (ITGA3) plasma membrane (ITGA3)
Anatomy Link Frequency
muscle 1
SCA 1
ITGA3 (Homo sapiens)
Pain Link Frequency Relevance Heat
cva 4 99.88 Very High Very High Very High
algogenic 12 99.02 Very High Very High Very High
bradykinin 4 97.32 Very High Very High Very High
spinothalamic 4 96.96 Very High Very High Very High
abdominal pain 1 84.80 Quite High
corticosteroid 2 77.12 Quite High
cytokine 13 5.00 Very Low Very Low Very Low
iatrogenic 4 5.00 Very Low Very Low Very Low
chemokine 4 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Epstein-barr Virus 51 100.00 Very High Very High Very High
Cytomegalovirus Infection 6 100.00 Very High Very High Very High
Cv General 3 Under Development 4 99.88 Very High Very High Very High
Hepatitis C Virus Infection 7 98.96 Very High Very High Very High
Hepatitis A Virus Infection 1 98.26 Very High Very High Very High
Infection 172 95.72 Very High Very High Very High
Liver Failure 1 92.24 High High
Splenomegaly 1 88.92 High High
Anaemia 1 88.24 High High
Hepatomegaly 1 87.92 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Therefore, in addition to spinothalamic convergence of somatic and visceral afferents, activation of SCA to generate spasmlike muscle contractions could account in part for anginal pain, and VCA activation could attenuate this effect.
Positive_regulation (activation) of VCA in muscle associated with cva and spinothalamic
1) Confidence 0.06 Published 2001 Journal Am. J. Physiol. Regul. Integr. Comp. Physiol. Section Abstract Doc Link 11705797 Disease Relevance 0.10 Pain Relevance 0.32
Activation with bradykinin (n = 46) produced a significantly higher peak response than infusion of an algogenic mixture (n = 53) containing chemicals that also activate VCA.
Positive_regulation (activate) of VCA associated with algogenic and bradykinin
2) Confidence 0.06 Published 2001 Journal Am. J. Physiol. Regul. Integr. Comp. Physiol. Section Abstract Doc Link 11705797 Disease Relevance 0 Pain Relevance 0.37
These results suggest an excitatory and inhibitory role for SCA and VCA, respectively.
Positive_regulation (role) of VCA in SCA
3) Confidence 0.04 Published 2001 Journal Am. J. Physiol. Regul. Integr. Comp. Physiol. Section Abstract Doc Link 11705797 Disease Relevance 0 Pain Relevance 0.35
Electrical stimulation of VCA did not evoke EMG activity but inhibited the chemically evoked EMG response (12 of 12).
Positive_regulation (stimulation) of VCA
4) Confidence 0.03 Published 2001 Journal Am. J. Physiol. Regul. Integr. Comp. Physiol. Section Abstract Doc Link 11705797 Disease Relevance 0 Pain Relevance 0.37
The IgM anti-VCA response disappears over the next few months but the IgG titer falls to a steady state after previously peaking.
Positive_regulation (response) of VCA
5) Confidence 0.02 Published 2005 Journal Virol J Section Body Doc Link PMC1243244 Disease Relevance 0.72 Pain Relevance 0
Anti-HAV IgM, HbsAg, anti-HBc IgM, anti-HCV and anti-CMV IgM were negative, while IgM VCA EBV, IgG VCA EBV and anti-CMV IgG were positive.
Positive_regulation (positive) of IgG VCA EBV associated with epstein-barr virus, cytomegalovirus infection, hepatitis c virus infection and hepatitis a virus infection
6) Confidence 0.00 Published 2002 Journal J. Infect. Section Abstract Doc Link 12217711 Disease Relevance 2.12 Pain Relevance 0.12

General Comments

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